CCAAT/enhancer binding protein zeta

CCAAT/enhancer binding protein (C/EBP), zeta
Identifiers
Symbols CEBPZ ; CBF; CBF2; HSP-CBF; NOC1
External IDs OMIM: 612828 MGI: 109386 HomoloGene: 4210 GeneCards: CEBPZ Gene
Orthologs
Species Human Mouse
Entrez 10153 12607
Ensembl ENSG00000115816 ENSMUSG00000024081
UniProt Q03701 P53569
RefSeq (mRNA) NM_005760 NM_001024806
RefSeq (protein) NP_005751 NP_001019977
Location (UCSC) Chr 2:
37.2 – 37.23 Mb
Chr 17:
78.92 – 78.94 Mb
PubMed search

CCAAT/enhancer-binding protein zeta is a protein that in humans is encoded by the CEBPZ gene.[1][2][3]

Interactions

CCAAT/enhancer binding protein zeta has been shown to interact with:

See also

References

  1. Lum LS, Sultzman LA, Kaufman RJ, Linzer DI, Wu BJ (Jan 1991). "A cloned human CCAAT-box-binding factor stimulates transcription from the human hsp70 promoter". Mol Cell Biol 10 (12): 6709–17. PMC 362949. PMID 2247079.
  2. 1 2 Uramoto H, Izumi H, Nagatani G, Ohmori H, Nagasue N, Ise T, Yoshida T, Yasumoto K, Kohno K (Apr 2003). "Physical interaction of tumour suppressor p53/p73 with CCAAT-binding transcription factor 2 (CTF2) and differential regulation of human high-mobility group 1 (HMG1) gene expression". Biochem J 371 (Pt 2): 301–10. doi:10.1042/BJ20021646. PMC 1223307. PMID 12534345.
  3. "Entrez Gene: CEBPZ CCAAT/enhancer binding protein zeta".
  4. Imbriano C, Bolognese F, Gurtner A, Piaggio G, Mantovani R (Jul 2001). "HSP-CBF is an NF-Y-dependent coactivator of the heat shock promoters CCAAT boxes". J. Biol. Chem. 276 (28): 26332–9. doi:10.1074/jbc.M101553200. PMID 11306579.
  5. Uramoto H, Izumi H, Nagatani G, Ohmori H, Nagasue N, Ise T, Yoshida T, Yasumoto K, Kohno K (Apr 2003). "Physical interaction of tumour suppressor p53/p73 with CCAAT-binding transcription factor 2 (CTF2) and differential regulation of human high-mobility group 1 (HMG1) gene expression". Biochem. J. 371 (Pt 2): 301–10. doi:10.1042/BJ20021646. PMC 1223307. PMID 12534345.

Further reading

External links

This article incorporates text from the United States National Library of Medicine, which is in the public domain.


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